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	Anti-neuroinflammatory effects of 20C from Gastrodia elata via regulating autophagy in LPS-activated BV-2 cells through MAPKs and TLR4/Akt/mTOR signaling pathways
	Shao, Qian-hang; Zhang, Xiao-ling; Chen, Ying; Zhu, Cheng-Gen; Shi, Jian-Gong; Yuan, Yu-he*; Chen, Nai-hong*
刊名	Molecular Immunology
	2018
卷号	99页码:115-123
关键词	Akt protein kinase B Atg autophagy related gene CNS central nervous system COX-2 cyclooxygenase-2 CQ chloroquine ERK extracellular signal-regulated kinase IL-1β interleukin-1β iNOS inducible nitric oxide synthase JNK c-jun NH2-terminal kinase LPS Lipopolysaccharide MAPKs mitogen-activated protein kinases mTOR mechanistic target of rapamycin NO nitric oxide PI3K phosphatidylinositol 3-kinase PRR pattern recognition receptor TLR4 toll like receptor 4 TNF-α tumor necrosis factor-α 20C Autophagy Mitogen-activated protein kinases (MAPKs) Toll like receptor 4 (TLR4) Protein kinase B (Akt) Mechanistic target of rapamycin (mTOR)
ISSN号	0161-5890
DOI	10.1016/j.molimm.2018.04.014
URL标识	查看原文
WOS记录号	WOS:000437042200013
内容类型	期刊论文
URI标识	http://www.corc.org.cn/handle/1471x/5706743
专题	湖南中医药大学
作者单位	1.[Shao, Qian-hang 2.Chen, Ying 3.Shi, Jian-Gong 4.Chen, Nai-hong 5.Zhang, Xiao-ling 6.Yuan, Yu-he 7.Zhu, Cheng-Gen] Chinese Acad Med Sci, Inst Mat Med, State Key Lab Bioact Subst & Funct Nat Med, Beijing 100050, Peoples R China.
推荐引用方式 GB/T 7714	Shao, Qian-hang,Zhang, Xiao-ling,Chen, Ying,et al. Anti-neuroinflammatory effects of 20C from Gastrodia elata via regulating autophagy in LPS-activated BV-2 cells through MAPKs and TLR4/Akt/mTOR signaling pathways[J]. Molecular Immunology,2018,99:115-123.
APA	Shao, Qian-hang.,Zhang, Xiao-ling.,Chen, Ying.,Zhu, Cheng-Gen.,Shi, Jian-Gong.,...&Chen, Nai-hong*.(2018).Anti-neuroinflammatory effects of 20C from Gastrodia elata via regulating autophagy in LPS-activated BV-2 cells through MAPKs and TLR4/Akt/mTOR signaling pathways.Molecular Immunology,99,115-123.
MLA	Shao, Qian-hang,et al."Anti-neuroinflammatory effects of 20C from Gastrodia elata via regulating autophagy in LPS-activated BV-2 cells through MAPKs and TLR4/Akt/mTOR signaling pathways".Molecular Immunology 99(2018):115-123.

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