Thyroid hormone protects cardiomyocytes from H2O2-induced oxidative stress via the PI3K-AKT signaling pathway | |
Zeng, Bin; Liu, Lei; Liao, Xiaoting; Zhang, Caixia; Ruan, Huaiyu | |
刊名 | EXPERIMENTAL CELL RESEARCH |
2019 | |
卷号 | 380期号:2 |
关键词 | Thyroid hormone Oxidative stress Apoptosis Mitochondrial function PI3K/AKT signaling |
ISSN号 | 0014-4827 |
DOI | 10.1016/j.yexcr.2019.05.003 |
URL标识 | 查看原文 |
收录类别 | SCIE |
语种 | 英语 |
内容类型 | 期刊论文 |
URI标识 | http://www.corc.org.cn/handle/1471x/4263836 |
专题 | 武汉大学 |
推荐引用方式 GB/T 7714 | Zeng, Bin,Liu, Lei,Liao, Xiaoting,et al. Thyroid hormone protects cardiomyocytes from H2O2-induced oxidative stress via the PI3K-AKT signaling pathway[J]. EXPERIMENTAL CELL RESEARCH,2019,380(2). |
APA | Zeng, Bin,Liu, Lei,Liao, Xiaoting,Zhang, Caixia,&Ruan, Huaiyu.(2019).Thyroid hormone protects cardiomyocytes from H2O2-induced oxidative stress via the PI3K-AKT signaling pathway.EXPERIMENTAL CELL RESEARCH,380(2). |
MLA | Zeng, Bin,et al."Thyroid hormone protects cardiomyocytes from H2O2-induced oxidative stress via the PI3K-AKT signaling pathway".EXPERIMENTAL CELL RESEARCH 380.2(2019). |
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