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Defects in MAP1S‐mediated autophagy cause reduction in mouse lifespans especially when fibronectin is overexpressed
Li, Wenjiao; Zou, Jing; Yue, Fei; Song, Kun; Chen, Qi; McKeehan, Wallace L.; Wang, Fen; Xu, Guibin; Huang, Hai*; Yi, Jinglin
刊名Aging Cell
2016
卷号15期号:2页码:370-379
关键词autophagy fibronectin lifespan liver fibrosis MAP1S
ISSN号1474-9726
DOI10.1111/acel.12441
URL标识查看原文
WOS记录号WOS:000372880500018
内容类型期刊论文
URI标识http://www.corc.org.cn/handle/1471x/3341712
专题中南大学
作者单位1.[Liu, Leyuan
2.Huang, Hai
3.Song, Kun
4.Xu, Guibin
5.Li, Wenjiao
6.Chen, Qi
7.Wang, Fen
8.Zou, Jing
9.Yue, Fei
10.McKeehan, Wallace L.] Texas A&M Hlth Sci Ctr, Inst Biosci & Technol, Ctr Translat Canc Res, 2121 W Holcombe Blvd, Houston, TX 77030 USA.
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Li, Wenjiao,Zou, Jing,Yue, Fei,et al. Defects in MAP1S‐mediated autophagy cause reduction in mouse lifespans especially when fibronectin is overexpressed[J]. Aging Cell,2016,15(2):370-379.
APA Li, Wenjiao.,Zou, Jing.,Yue, Fei.,Song, Kun.,Chen, Qi.,...&Liu, Leyuan*.(2016).Defects in MAP1S‐mediated autophagy cause reduction in mouse lifespans especially when fibronectin is overexpressed.Aging Cell,15(2),370-379.
MLA Li, Wenjiao,et al."Defects in MAP1S‐mediated autophagy cause reduction in mouse lifespans especially when fibronectin is overexpressed".Aging Cell 15.2(2016):370-379.
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