Neuroprotection induced by sevoflurane-delayed post-conditioning is attributable to increased phosphorylation of mitochondrial GSK-3β through the PI3K/Akt survival pathway | |
Ye, Zhi; Xia, Pingping; Cheng, Zhi-gang; Guo, Qulian* | |
刊名 | Journal of the Neurological Sciences |
2015 | |
卷号 | 348期号:1-2页码:216-225 |
关键词 | Apoptosis Focal cerebral ischemia Glycogen synthase kinase 3beta (GSK-3beta) PI3K/Akt Post-conditioning Sevoflurane |
ISSN号 | 0022-510X |
DOI | 10.1016/j.jns.2014.12.011 |
URL标识 | 查看原文 |
WOS记录号 | WOS:000349588300033 |
内容类型 | 期刊论文 |
URI标识 | http://www.corc.org.cn/handle/1471x/3333478 |
专题 | 中南大学 |
作者单位 | 1.[Guo, Qulian 2.Xia, Pingping 3.Cheng, Zhi-gang 4.Ye, Zhi] Affiliated Xiangya Hosp Cent South Univ, Dept Anesthesiol, Changsha 410078, Hunan, Peoples R China. |
推荐引用方式 GB/T 7714 | Ye, Zhi,Xia, Pingping,Cheng, Zhi-gang,et al. Neuroprotection induced by sevoflurane-delayed post-conditioning is attributable to increased phosphorylation of mitochondrial GSK-3β through the PI3K/Akt survival pathway[J]. Journal of the Neurological Sciences,2015,348(1-2):216-225. |
APA | Ye, Zhi,Xia, Pingping,Cheng, Zhi-gang,&Guo, Qulian*.(2015).Neuroprotection induced by sevoflurane-delayed post-conditioning is attributable to increased phosphorylation of mitochondrial GSK-3β through the PI3K/Akt survival pathway.Journal of the Neurological Sciences,348(1-2),216-225. |
MLA | Ye, Zhi,et al."Neuroprotection induced by sevoflurane-delayed post-conditioning is attributable to increased phosphorylation of mitochondrial GSK-3β through the PI3K/Akt survival pathway".Journal of the Neurological Sciences 348.1-2(2015):216-225. |
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