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Hes1 triggers epithelial-mesenchymal transition (EMT)-like cellular marker alterations and promotes invasion and metastasis of nasopharyngeal carcinoma by activating the PTEN/AKT pathway
Wang, Sheng-Chun[1,5]; Lin, Xiao-Lin[1]; Wang, Hui-Yan[1]; Qin, Yu-Juan[1]; Chen, Lin[1]; Li, Jing[1]; Jia, Jun-Shuang[1]; Shen, Hong-Fen[1]; Yang, Sheng[1]; Xie, Rao-Ying[1]
2015
卷号6期号:[db:dc_citation_issue]页码:36713
DOI[db:dc_identifier_doi]
URL标识查看原文
WOS记录号[DB:DC_IDENTIFIER_WOSID]
内容类型期刊论文
URI标识http://www.corc.org.cn/handle/1471x/3333308
专题暨南大学
作者单位1.[1]Southern Med Univ, Canc Res Inst, Guangzhou 510515, Guangdong, Peoples R China
2.[2]Southern Med Univ, Inst Comparat Med, Guangzhou 510515, Guangdong, Peoples R China
3.[3]Southern Med Univ, Lab Anim Ctr, Guangzhou 510515, Guangdong, Peoples R China
4.[4]Harvard Univ, Sch Med, Childrens Hosp Boston, Joint Program Transfus Med, Boston, MA 02115 USA
5.[5]Guangdong Med Univ, Dept Pathol, Dongguan 523808, Peoples R China
6.[6]Southern Med Univ, Tradit Chinese Med Integrated Hosp, Dept Oncol, Guangzhou 510315, Guangdong, Peoples R China
7.[7]Jinan Univ, Affiliated Hosp 1, Dept Gastroenterol, Guangzhou 510630, Guangdong, Peoples R China
推荐引用方式
GB/T 7714
Wang, Sheng-Chun[1,5],Lin, Xiao-Lin[1],Wang, Hui-Yan[1],et al. Hes1 triggers epithelial-mesenchymal transition (EMT)-like cellular marker alterations and promotes invasion and metastasis of nasopharyngeal carcinoma by activating the PTEN/AKT pathway[J],2015,6([db:dc_citation_issue]):36713.
APA Wang, Sheng-Chun[1,5].,Lin, Xiao-Lin[1].,Wang, Hui-Yan[1].,Qin, Yu-Juan[1].,Chen, Lin[1].,...&Xiao, Dong[1,2,3].(2015).Hes1 triggers epithelial-mesenchymal transition (EMT)-like cellular marker alterations and promotes invasion and metastasis of nasopharyngeal carcinoma by activating the PTEN/AKT pathway.,6([db:dc_citation_issue]),36713.
MLA Wang, Sheng-Chun[1,5],et al."Hes1 triggers epithelial-mesenchymal transition (EMT)-like cellular marker alterations and promotes invasion and metastasis of nasopharyngeal carcinoma by activating the PTEN/AKT pathway".6.[db:dc_citation_issue](2015):36713.
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