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Glutamate oxidative injury to RGC-5 cells in culture is necrostatin sensitive and blunted by a hydrogen sulfide (H2S)-releasing derivative of aspirin (ACS14)
Neville N. Osborne; Dan Ji; Aman Shah Abdul Majid; Piero Del Soldata; Anna Sparatore
刊名Neurochemistry International
2012
卷号60期号:4页码:365-378
关键词Aspirin ACS14 Hydrogen sulphide Glutamate toxicity Oxidative stress Neuroprotection Necrostatin
ISSN号0197-0186
DOI10.1016/j.neuint.2012.01.015
URL标识查看原文
WOS记录号WOS:000302510900006
内容类型期刊论文
URI标识http://www.corc.org.cn/handle/1471x/3319030
专题中南大学
作者单位1.[Neville N. Osborne
2.Dan Ji
3.Aman Shah Abdul Majid] Univ Oxford, John Radcliffe Hosp, Nuffield Lab Ophthalmol, Level 6,West Wing,Headley Way, Oxford OX3 9DU, England.
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Neville N. Osborne,Dan Ji,Aman Shah Abdul Majid,et al. Glutamate oxidative injury to RGC-5 cells in culture is necrostatin sensitive and blunted by a hydrogen sulfide (H2S)-releasing derivative of aspirin (ACS14)[J]. Neurochemistry International,2012,60(4):365-378.
APA Neville N. Osborne,Dan Ji,Aman Shah Abdul Majid,Piero Del Soldata,&Anna Sparatore.(2012).Glutamate oxidative injury to RGC-5 cells in culture is necrostatin sensitive and blunted by a hydrogen sulfide (H2S)-releasing derivative of aspirin (ACS14).Neurochemistry International,60(4),365-378.
MLA Neville N. Osborne,et al."Glutamate oxidative injury to RGC-5 cells in culture is necrostatin sensitive and blunted by a hydrogen sulfide (H2S)-releasing derivative of aspirin (ACS14)".Neurochemistry International 60.4(2012):365-378.
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