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	BmP02 Atypically Delays Kv4.2 Inactivation: Implication for a Unique Interaction between Scorpion Toxin and Potassium Channel
	Wu, Bin[1]; Zhu, Yan[2]; Shi, Jian[3]; Tao, Jie[4]; Ji, Yonghua[5]
刊名	TOXINS
	2016
卷号	8
关键词	scorpion toxin voltage-gated potassium channels electrophysiology interaction mode
ISSN号	2072-6651
URL标识	查看原文
内容类型	期刊论文
URI标识	http://www.corc.org.cn/handle/1471x/2230802
专题	上海大学
作者单位	1.[1]Shanghai Univ, Lab Neuropharmacol & Neurotoxicol, Nanchen Rd 333, Shanghai 200444, Peoples R China. 2.[2]Shanghai Univ, Lab Neuropharmacol & Neurotoxicol, Nanchen Rd 333, Shanghai 200444, Peoples R China. 3.[3]Shanghai Univ, Lab Neuropharmacol & Neurotoxicol, Nanchen Rd 333, Shanghai 200444, Peoples R China. 4.St Georges Univ London, Vasc Biol Res Ctr, Inst Cardiovasc & Cell Sci, Cranmer Terrace, London SW17 0RE, England. 5.[4]Shanghai Univ Tradit Chinese Med, Putuo Hosp, Cent Lab, 164 Lanxi Rd, Shanghai 200062, Peoples R China. 6.[5]Shanghai Univ, Lab Neuropharmacol & Neurotoxicol, Nanchen Rd 333, Shanghai 200444, Peoples R China.
推荐引用方式 GB/T 7714	Wu, Bin[1],Zhu, Yan[2],Shi, Jian[3],et al. BmP02 Atypically Delays Kv4.2 Inactivation: Implication for a Unique Interaction between Scorpion Toxin and Potassium Channel[J]. TOXINS,2016,8.
APA	Wu, Bin[1],Zhu, Yan[2],Shi, Jian[3],Tao, Jie[4],&Ji, Yonghua[5].(2016).BmP02 Atypically Delays Kv4.2 Inactivation: Implication for a Unique Interaction between Scorpion Toxin and Potassium Channel.TOXINS,8.
MLA	Wu, Bin[1],et al."BmP02 Atypically Delays Kv4.2 Inactivation: Implication for a Unique Interaction between Scorpion Toxin and Potassium Channel".TOXINS 8(2016).

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