QQ客服 官方微博 反馈留言

	miR-217 targeting DKK1 promotes cancer stem cell properties via activation of the Wnt signaling pathway in hepatocellular carcinoma
	Jiang, Chunlin[1,2]; Yu, Miao[3]; Xie, Xiaoyan[1,2]; Huang, Guangliang[1,2]; Peng, Yao[5]; Ren, Dong[6]; Lin, Manxia[1,2]; Liu, Baoxian[1,2]; Liu, Ming[1,2]; Wang, Wei[1]
刊名	ONCOLOGY REPORTS
	2017
卷号	38页码:2351-2359
关键词	miR-217 cancer stem cells DKK1 Wnt signaling pathway hepatocellular carcinoma
URL标识	查看原文
内容类型	期刊论文
URI标识	http://www.corc.org.cn/handle/1471x/2182498
专题	华南理工大学
作者单位	1.[1]Sun Yat Sen Univ, Affiliated Hosp 1, Inst Diagnost & Intervent Ultrasound, Dept Med Ultrason, Guangzhou 510080, Guangdong, Peoples R China 2.[2]Sun Yat Sen Univ, Affiliated Hosp 1, Div Intervent Ultrasound, Guangzhou 510080, Guangdong, Peoples R China 3.[3]Sun Yat Sen Univ, Affiliated Hosp 1, Ctr Private Med Serv & Healthcare, Guangzhou 510080, Guangdong, Peoples R China 4.[4]Sun Yat Sen Univ, Affiliated Hosp 1, Dept Liver Surg, Orthopaed Res Inst, Guangzhou 510080, Guangdong, Peoples R China 5.[5]Sun Yat Sen Univ, Affiliated Hosp 1, Dept Gastroenterol, Orthopaed Res Inst, Guangzhou 510080, Guangdong, Peoples R China 6.[6]Sun Yat Sen Univ, Affiliated Hosp 1, Dept Orthopaed Surg, Orthopaed Res Inst, Guangzhou 510080, Guangdong, Peoples R China
推荐引用方式 GB/T 7714	Jiang, Chunlin[1,2],Yu, Miao[3],Xie, Xiaoyan[1,2],et al. miR-217 targeting DKK1 promotes cancer stem cell properties via activation of the Wnt signaling pathway in hepatocellular carcinoma[J]. ONCOLOGY REPORTS,2017,38:2351-2359.
APA	Jiang, Chunlin[1,2].,Yu, Miao[3].,Xie, Xiaoyan[1,2].,Huang, Guangliang[1,2].,Peng, Yao[5].,...&Kuang, Ming[2,4].(2017).miR-217 targeting DKK1 promotes cancer stem cell properties via activation of the Wnt signaling pathway in hepatocellular carcinoma.ONCOLOGY REPORTS,38,2351-2359.
MLA	Jiang, Chunlin[1,2],et al."miR-217 targeting DKK1 promotes cancer stem cell properties via activation of the Wnt signaling pathway in hepatocellular carcinoma".ONCOLOGY REPORTS 38(2017):2351-2359.

个性服务

查看访问统计

相关权益政策

暂无数据

收藏/分享

除非特别说明，本系统中所有内容都受版权保护，并保留所有权利。